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The people who coined the terms filthy lucre and dirty money may have been on to something.
Swiss researchers have reported that influenza viruses can survive - alive and potentially infectious - on bank notes for up to 17 days in some cases.
It’s not known what portion of influenza transmission is due to the touching of contaminated surfaces with hands which then transport viruses to the vulnerable mucous membranes of the nose or mouth. And this study can’t answer that question.
But lead author Yves Thomas said Wednesday he believes the touching of contaminated surfaces plays a role in the spread of flu. And those contaminated surfaces can include folding money.
“When you see that the virus is still alive for several days, I can’t imagine that it does not infect. I’m sure that it can infect,” Thomas, a virologist at the Swiss National Centre for Influenza, said at a major international conference, Options for the Control of Influenza.
“It’s still alive. And it’s alive in quantities that can infect.”
But a Toronto-based infectious diseases expert said she isn’t convinced.
“The problem with all of the environmental studies of influenza and other pathogens is the fact that these bacteria and viruses survive in the environment doesn’t mean they are transmitted by the environment,” said Dr. Allison McGeer, head of infection control at Toronto’s Mount Sinai Hospital.
“And while it sounds just counterintuitive to say that if the virus is there in the environment you can’t catch it, there is in fact a substantial amount of epidemiological evidence that that’s true.”
McGeer said, for instance, that there’s really no proof people who handle bank notes for a living suffer more bouts of the flu than those who don’t.
“Now people will argue that maybe we haven’t looked for it,” she acknowledged.
“But the truth of the matter is that kids don’t handle bank notes. But there’s lots of evidence that infection rates are highest in children and children transmit the most. And influenza was transmitted pretty regularly before we had bank notes.”
The work was done at the behest of Swiss National Bank, which provided the currency on which the testing was done.
Rising concern about the possibility of a flu pandemic had executives worried that bank notes might serve as a virus delivery system, both for bank employees who handle paper money, and for the broader public.
So the scientists inoculated small pieces of bills with several different strains of human influenza - an influenza B virus and three influenza A viruses. Two of the As were H3N2 viruses and the third was an H1N1.
They allowed the notes to dry naturally, and kept them at room temperature (22 C). At various points they submerged pieces of the notes in a medium, then tried to see if the medium contained live viruses by putting it into culture to see if virus would grow.
The H1N1 and influenza B viruses didn’t last long on the money, dying within an hour or so. But one of the H3N2 viruses survived about 24 hours and another was viable up to 72 hours, Thomas said.
The team then decided to try to see if mucus would enhance survival. They mixed the viruses with mucus and swabbed a thin slick of the stuff on bank notes.
The difference in survival time was substantial. “With secretory mucus, it’s 17 days,” Thomas said.
As with the first round of the work, the survival time varied depending on the subtype of virus, with 17 days being the longest period for which virus was viable. The influenza B virus, for instance, lasted 24 hours in mucus but only two hours without it.
The mucus may keep the virus from drying out, Thomas suggested, noting another scientist ventured the view that the protein in the mucus may play some role in the extended survival time.
But laboratory experiments, done under controlled conditions, don’t always reflect what happens in the real world.
So the group tried a third phase of the work, swabbing still more bills with “nasopharyngeal secretions” - yes, that’s snot - from 14 flu-infected children.
At 24 hours, live viruses were retrieved from 50 per cent of the bills. At 48 hours, there was live virus found on 30 per cent.
Thomas said he believes that depending on the virus used, the survival in mucus ranges from about two to 17 days, and is probably closer to the lower end of that spectrum

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three optimal lengths of sleep -­ but that doesn’t mean you can just choose one. A study completed this spring by Washington State University Spokane suggests that our sleep patterns are embedded in our bodies - perhaps in our very genes. Some of us will need five-and-a-half hours of sleep, while others will need at least eight-and-a-half. Most people will manage comfortably on seven hours. Your genes decide for you and you can’t just alter it without paying the price.

There is hardly anybody out there who knows what it means to be fully awake. Studies have found that proper sleeping patterns emerge only after you have caught up with up to 25 hours of missing sleep. To optimize your sleep, crawl into bed half an hour earlier each evening for a few nights. As long as you have a sleep deficit to catch up on, you should have no problem falling asleep. After that, allow yourself as much sleep as you need. If you persistently sleep too little, you run the risk of becoming overweight, absent-minded and ill; a daily sleep deficit of two hours over a period of 14 days is as damaging as a night with no sleep.

Sleeping too much is also a rest buster. If you sleep for longer than your personal optimal period, your sleep will be empty and restless. If you oversleep for many hours, you will fall into another deep sleep in the morning. This will upset your circadian clock and you will wake up feeling absolutely whacked. If this is your problem, you can reverse the situation by keeping your time in bed to the absolute minimum and staying up a bit later at night to prolong the restful deep sleep at the beginning of the night.

3. Worship the sun.

Most people can get away with some wildness in their routines as long as they soak up some bright light at the right time. Normal indoor lighting provides 400 lux of illumination, which doesn’t help much; the sun, however, provides 1 500 to 100 000 lux. So if you spend one hour outdoors before starting work you will be more alert and cheerful during the day.

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Antibiotic use had a greater impact on children who would otherwise be considered at lower risk — children who lived in rural areas and those whose mothers did not have asthma — than on those who were already at increased risk because of an urban environment or genetic predisposition.

Studies of antibiotic use and asthma have been complicated. Because antibiotics are used to treat respiratory illnesses, which are often precursors of asthma, it has been difficult to determine the effect of antibiotics alone. But this study, of 13,116 Canadian children, found that the risk of asthma increased even in children treated with antibiotics for nonrespiratory illnesses in the first year of life. The study appears in the June issue of Chest.

Anita Kozyrskyj, the lead author and an associate professor of pharmacy at the University of Manitoba, said the findings supported what scientists call the microflora hypothesis — that “you need good bacteria in your digestive tract for normal development of the immune system so that you don’t end up with asthma,” as she put it.

The researchers tracked medications by examining prescription records, and determined asthma status by treatment for asthma or any asthma drug use in the year following the seventh birthday. Six percent of the children developed asthma by age 7.

After statistically adjusting for respiratory and nonrespiratory illnesses, sex, maternal history of asthma, urban or rural location and other factors, researchers found that one or two courses of antibiotics in the first year of life increased the risk of asthma by about 20 percent.

The more frequent the antibiotic use, the higher the risk. Three to four courses of medicine conferred a 30 percent added risk, and more than four courses of antibiotics increased the risk by almost 50 percent.

The findings were stronger for the use of broad-spectrum antibiotics like the cephalosporins and amoxicillin than they were for narrow-spectrum drugs like penicillin and erythromycin. “Lactobacilli, for example, are more affected by the broad-spectrum drugs,” Dr. Kozyrskyj said, referring to the beneficial bacteria contained in yogurt.

In a secondary finding using a small part of the sample population, researchers found that among children who had multiple courses of antibiotics in infancy, those who lived with no dog in the house had twice the risk of asthma compared with those who lived with one. The reasons are not clear, but it may be that having less contact with the germs that dogs carry results in lower microbial loads, making a child more sensitive to antibiotics. Dr. Kozyrskyj said in an interview that she found no decreased risk with the presence of cats or other pets.

Jeroen Douwes, a professor of epidemiology at Massey University in New Zealand who has published widely on asthma in children but was not involved in this work, cited the study’s strong methodology. “They had very good data on antibiotic use during the first year of life, and that’s actually quite rare,” he said. “They measured exposure before the disease occurred, while in most studies you have to rely on people’s recollections.”

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If you have an allergy to something specific ( a food , chemical or inhalant) for example , it is common to develop allergies to other substances, especially if you have a high level of exposure to them. This pre disposing to develop further allergies is well documented and runs in families. Some people with a specific allergy never develop an allergy to anything else but many and most allergy sufferers do.

People suffering from chemical sensitivity and food intolerance which are disorders in which your immune and body systems react to chemicals and foods sometimes without even involving the very natural immune system. These individuals develop very widespread sensivity to all kinds of things , which seem to aggrevate each other. It is a syneristic attack. The whole is more than the parts. In some cases , people appear to be sensitive to almost everything around them ( allergic disease the boy in the bubble) .

This type of reaction , a severe immune reaction , has been given all types of names and markers including “multiple sensitivity ” , ” multiple allergic sensitivities” and “total allergy sydrome”.

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Allergies as with most problems in life often come in multiples. If you are lucky or unlucky enough to be sensitive in your immune system to have one allergy it is like viruses and parasites in computers and computer science then you most likely are destined to have more.

Why is it that some people have this tendency to develop multiple allergies and yet others never seem to develop this medical and health trend ?

In either case you and your health concerns will be paramount to your functions in daily life and living.

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There is much controversy over what an allergy actually is . Historically the definition is very broad encompassing a wide range of illnesses brought about by people working with substances in their environment. By the late 1960’s scientific research into the workings and underpinnings of the immune system ( immunology - later a key science int the understanding and comprehension of the aids virus or non virus worldwide illness pandemic) had identified the mechanisms underlying most of the classic allergic diseases- asthma, eczema , hay fever , perennial rhinitis , uticaria , anaphylactic shop ( anaphalysis) .

As time went on these allergic disorders served as the basic models for understanding of the allergic response and the role played in many mammalian disease and disease states.

The role of allergies in the pathology of many diseases such as rheumatoid arthritis and mental illness became to be more and more appreciated.

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Allergy means an abnormal excessive reaction of the body’s defence systems to a substance absorbed from the environment . Allergies now affect a high and higher percentage and are well known to be on the continual increase in the general population. Hayfever , asthma etc etc climb in incidence higher and higher. More people suffer. More

people will tell you that they suffer from allergies - cannot eat this or that because of allergies , are taking antihistamines , inhalers for their asthma , antibiotics for infections , pills and tablets and salves. Dermatologists abound . And you wonder why or why not ??? Why has the health of the general population and your health as a human being been on the medical and tentorial downslide ???

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Symptoms

To date, no clinical studies have looked into Morgellons and only one paper mentioning Morgellons has been published in a medical journal. Appearing in a recent issue of the American Journal of Clinical Dermatology, the paper is co-authored by members of the Morgellons Research Foundation, a non-profit organization devoted to raising public awareness about the disease.

As of February 2006, more than 2,000 reports of the disease have been reported on the Foundation’s website. Reports come from all 50 U.S. states and 15 nations, including Canada, the UK, Australia and The Netherlands.

The majority of reports have come from Texas, California and Florida.

Patients with the disease often describe feelings of insects or parasites scuttling beneath their skin and open lesions that heal slowly and which ooze out blue and white fibers, some as thick as spaghetti strands. Attempts to remove the fibers are said to elicit shooting pains radiating from the site.

The lesions range from minor to disfiguring in appearance and fibers appear either as single strands or as bundles. Patients also sometimes report the presence of fibers or black granular specks on their skin even in the absence of lesions. Some patients even report symptoms of the disease in their pets–dogs mostly, but also cats and horses.

According to statistics from the Morgellons Research Foundation, about 95 percent of patients also report suffering from disabling fatigue, or “brain fog,” that hinders their ability to pay attention. Other reported symptoms include joint pain, sleep disorders, hair loss, decline in vision, and even the “disintegration” of perfectly healthy teeth. It appears that once patients contract the disease, they have it for life. To date, there have been no reports of spontaneous remissions.

Strange fibers

A preliminary analysis of the fibers suggests they are more than just lint from household materials such as clothing, carpets or bedding, said Randy Wymore, an assistant professor of pharmacology and physiology at Okalahoma State University and the director of research at the Morgellons Research Foundation.

“The fibers are not common textiles, nor are they black specks of pepper, as several dermatologists have proposed,” Wymore told LiveScience.

Further deepening the mystery, some analyses suggest the fibers might be made of cellulose, a molecule generally found in plants.

“They’re basically fibers that you wouldn’t expect to see in humans,” said Raphael Stricker, a Lyme disease expert at the California Pacific Medical Center in San Francisco and a medical advisor to the Morgellons Research Foundation.

The disease is named after a medical condition described in 1674 by the British author Thomas Browne. Known as “Morgellons,” Browne said the disorder caused children to “critically break out with harsh hairs on their backs…” The Morgellons Research Foundation says that it is doubtful that the 17^th century disease is related in any way to modern day Morgellons.

Skepticism

Despite increasing reports of the condition, many doctors have barely heard of the disease and many treat it with a heavy dose of skepticism.

Michael Giradi, a dermatologist at the Yale School of Medicine, had never heard of Morgellons but when its symptoms were described to him, he was reminded of another disorder that is well known to doctors.

“They just renamed it,” Giradi told LiveScience. “We just call it delusions of parasitosis.”

Also known as Eckbom syndrome, delusional parasitosis is a psychiatric disorder in which patients fervently believe their bodies are infected by skin parasites that do not exist.

“It’s basically when a patient thinks that there’s something coming out of their skin, a material or bug of some sort, when truthfully there’s nothing there,” said Stacy Beaty, a dermatologist at the Saint Louis University School of Medicine.

In medical schools, physicians learn to watch out for the “matchbox sign” of delusional parasitosis, when patients bring in hair, skin or clothing lint, sometimes in matchboxes, that they claim contain the insects or parasites responsible for their torment. However, when examined, the samples reveal no such thing. The lesions and scratches sometimes seen on patients with delusional parasitosis are usually self-inflicted, Beaty said.

“To rule out any infectious causes and also to put the patient’s mind at ease, a lot of times we’ll do skin biopsies,” Beaty said in a telephone interview. “If we feel that it’ll be helpful, we might also start different anti-psychotic or anti-anxiety medicines.”

Beaty said she was vaguely familiar with reports of Morgellons disease, but said that other doctors she had queried had never even heard of it.

In response to rising media coverage about the condition, the Los Angeles Department of Health Services recently issued a statement that said bluntly:

“No credible medical or public health association has verified the existence or diagnosis of ‘Morgellons Disease.’ The current description of the disease is vague and covers many conditions.”

Wymore, the Oklahoma State University researcher, says that debating whether Morgellons is a real disease or not is not the right approach.

“This population is suffering greatly,” Wymore said. “A better question would be, ‘Is Morgellons Disease a purely psychiatric disorder?’ and the answer is ‘No.’ Morgellons also has physical effects on a person. In addition to the skin lesions and the unusual fibers and other shed material, there are nervous system effects that include behavioral changes, cognitive changes and peripheral neuropathy.

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